We have a suspicion that you are an automated web bot software, not a real user. To keep our site fast for other users, we have slowed down this page. The slowdown will gradually disappear. If you think this is a mistake, please contact us at genome-www@soe.ucsc.edu. Also note that all data for hgGeneGraph can be obtained through our public MySQL server and all our software source code is available and can be installed locally onto your own computer. If you are unsure how to use these resources, do not hesitate to contact us.
UCSC Genome Browser Gene Interaction Graph
Gene interactions and pathways from curated databases and text-mining

◀ Back to IL10

IL10 — NOX1

Text-mined interactions from Literome

Tojo et al., J Hypertens 2005 (Hypertension, Renal) : Nicotinamide adenine dinucleotide phosphate ( NAD ( P ) H ) oxidase is regulated by angiotensin II, interleukin (IL)-1beta and tumor necrosis factor (TNF)-alpha via p38 mitogen activated protein kinase ( MAPK )
Perianayagam et al., Eur J Clin Invest 2005 (Uremia) : In contrast, apocynin, a selective NADPH oxidase inhibitor, did not inhibit ET-stimulated IL-10 production
Dang et al., FASEB J 2006 : This IL-10 effect could contribute to the tight regulation of NADPH oxidase activity at the inflammatory site
Kassan et al., Arterioscler Thromb Vasc Biol 2011 (Disease Models, Animal...) : These data demonstrate that IL-10 released from Tregs attenuates NADPH oxidase activity, which is a critical process in the improvement of microvascular endothelial function in hypertension, suggesting that Tregs/IL-10 could be a therapeutic target for treatment of vasculopathy in hypertension
Dammanahalli et al., J Hypertens 2011 : IL-10KO increased Nox1 protein expression, NADPH oxidase activity, and superoxide production in aortas