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CSF2 — NM

Text-mined interactions from Literome

Gomez-Cambronero et al., FEBS Lett 2003 : Rapamycin inhibits GM-CSF induced neutrophil migration
Shima et al., Am J Hypertens 2008 : GM-CSF induced neutrophil migration was also suppressed by amlodipine and nicardipine, but not by azelnidipine, when these blockers were assessed for their effect on neutrophil migration
Khajah et al., J Leukoc Biol 2011 : GM-CSF induced neutrophil migration was ablated ( < 95 % ) using neutrophils derived from GMCSFRß ( -/- ) mice and significantly attenuated by 42 % in PI3K? ( -/- ) neutrophils
Kariya et al., Laryngoscope 2013 (Disease Models, Animal...) : We hypothesized that granulocyte/macrophage colony stimulating factor plays a pivotal role in middle ear inflammation and that neutralization of granulocyte/macrophage colony stimulating factor would inhibit neutrophil migration into the middle ear and production of inflammatory mediators
Addison et al., Clin Exp Immunol 1989 : Granulocyte-macrophage colony stimulating factor may inhibit neutrophil migration in vivo
Yong et al., J Immunol 1993 : Granulocyte-macrophage-colony stimulating factor differentially regulates neutrophil migration across IL-1 activated and nonactivated human endothelium ... We demonstrate, using an in vitro model of the vascular endothelial barrier, that GM-CSF enhances neutrophil migration across unstimulated endothelium, increasing the percentage of migrating cells from 7.7 +/- 0.9 % ( mean +/- SE ) in controls to 12.5 +/- 1.5 % in the presence of GM-CSF ( 100 ng/ml ) ( n = 14, p < 0.0005 ) ... Hence GM-CSF acts to inhibit neutrophil migration across IL-1 activated endothelium, and almost completely abolishes IL-1 induced migration ( n = 7, p < 0.0005 ) ... This differential effect of GM-CSF on neutrophil migration , depending upon the conditions of endothelial activation, does not relate to an effect on adhesion, because GM-CSF has no effect on the increased adhesion of neutrophils to IL-1 treated endothelium ... The effect of GM-CSF on neutrophil migration across activated endothelium may be relevant to the clinical administration of human rGM-CSF
Yong et al., Br J Haematol 1996 : G-CSF differs from GM-CSF, however, in that although GM-CSF inhibited neutrophil migration across IL-1 activated endothelium ( 33 +/- 8 % inhibition, n = 6, P < 0.01 ), G-CSF had no effect on neutrophil migration across IL-1 activated endothelium
Metcalf et al., Blood 1996 (Bacterial Infections...) : The lack of involvement of either CSF in the neutrophil migration was confirmed by the development of typical neutrophil exudates when casein was injected into mice with inactivation of the genes encoding GM-CSF, G-CSF, or the beta-common chain of the GM-CSF receptor