◀ Back to CSF3

CASP2 — CSF3

Text-mined interactions from Literome

Suzuki et al., J Immunol 2001 (Inflammation) : The cleavage of both molecules was inhibited by G-CSF and benzyloxycarbonyl-Val-Ala-Asp-fluoromethylketone, a caspase inhibitor , both of which can inhibit neutrophil apoptosis
Schmidt-Mende et al., Leukemia 2001 (Anemia, Sideroblastic) : We showed that G-CSF significantly reduced Fas mediated caspase-8 and caspase-3-like activity and the degree of nuclear apoptotic changes in bone marrow from nine RARS patients ... This study suggests that G-CSF may reduce the pathologically increased caspase activity and concomitant apoptotic changes, and promote erythroid growth and differentiation of stem cells from RARS patients
Maianski et al., Blood 2002 : Granulocyte colony stimulating factor inhibits the mitochondria dependent activation of caspase-3 in neutrophils ... Yet, G-CSF prevented neither caspase-3 activation nor apoptosis in cytoplasts, confirming the need for protein synthesis in the G-CSF effects
Fan et al., Zhonghua Xue Ye Xue Za Zhi 2000 (Leukemia) : G-CSF induced apoptosis of leukemic cells through activation of caspase 3
Boneberg et al., Eur J Immunol 2002 : The conclusion that G-CSF inhibits caspase-1 activity was supported bythe finding that the release of IL-18 was also inhibited by G-CSF, similar to IL-1beta release ... G-CSF had no effect on LPS induced doubling of caspase-1 mRNA, indicating that G-CSF affects caspase-1 activation and not its formation
Tehranchi et al., Blood 2003 (Myelodysplastic Syndromes) : Moreover, we have previously reported that G-CSF inhibits Fas induced caspase activation in sideroblastic anemia ( RARS )
Huang et al., Neurobiol Aging 2007 : The 6-OHDA caused caspase-3 activation in dopaminergic neurons was inhibited by G-CSF