◀ Back to ICAM1
ICAM1 — TLR2
Text-mined interactions from Literome
Harokopakis et al., J Immunol 2006
:
Furthermore, fimbriae stimulated monocyte adhesion to HUVEC and transmigration across HUVEC monolayers ; both activities required
TLR2 and Rac1 signaling and were
dependent upon
ICAM-1 and the high-affinity state of CD11b/CD18
Boyd et al., Cardiovasc Res 2006
(Myocarditis) :
Ligand activation of TLR2,
TLR4 and TLR5, but not TLR3, TLR7 or TLR9,
resulted in cardiomyocyte expression of the inflammatory cytokine IL-6, the chemokines KC and MIP-2, and the cell surface adhesion molecule
ICAM-1 ... Ligand activation of
TLR2 , TLR4 and TLR5, but not TLR3, TLR7 or TLR9,
resulted in cardiomyocyte expression of the inflammatory cytokine IL-6, the chemokines KC and MIP-2, and the cell surface adhesion molecule
ICAM-1
Riccioli et al., J Immunol 2006
:
In addition,
TLR2/TLR6 and TLR5 stimulation
induces increased
ICAM-1 expression in Sertoli cells ... In addition,
TLR2/TLR6 and TLR5 stimulation
induces increased
ICAM-1 expression in Sertoli cells
Xie et al., Cell Microbiol 2008
(Otitis Media with Effusion) :
ICAM-1 upregulation on human monocytes by the LOS
required surface CD14,
TLR4 , NF-kappaB p65 and c-Jun N-terminal kinase (JNK) activity
Sawa et al., Cell Tissue Res 2008
:
The expression of LTA induced E-selectin,
ICAM-1 , and VCAM-1 in LEC is
suppressed by
anti-TLR2 but not by anti-TLR4 and is also suppressed by TLR2-specific short interfering RNA ( siRNA ) but not by siRNA for TLR4
Farina et al., Ann Rheum Dis 2011
(Scleroderma, Systemic) :
dsRNA/poly ( I:C ), but not other
TLR ligands, highly
stimulated ET-1 protein and mRNA ( EDN1 ), as well as
intercellular adhesion molecule-1 ( ICAM-1 ) and IFN regulated MX2, by endothelial cells and dermal fibroblasts
Li et al., Cardiovascular diabetology 2010
:
Stimulation of
TLR2 or TLR4
induced NF-?B activation, and the expression of
ICAM-1 , IL-6 and IL-8
Wilhelmsen et al., Innate Immun 2012
(Sepsis) :
We found that
TLR2 activation specifically
induces the expression of the genes IL-6, IL-8, CSF2, CSF3,
ICAM1 and SELE by human umbilical vein ECs and human lung microvascular ECs
Oarada et al., J Nutr Biochem 2013
(Inflammation) :
TLR2 deficiency significantly
attenuated the refeeding induced increase in the liver expression of Cxcl10, Cxcl1,
Icam-1 and A20