ID:AMPD3_HUMAN DESCRIPTION: RecName: Full=AMP deaminase 3; EC=3.5.4.6; AltName: Full=AMP deaminase isoform E; AltName: Full=Erythrocyte AMP deaminase; FUNCTION: AMP deaminase plays a critical role in energy metabolism. CATALYTIC ACTIVITY: AMP + H(2)O = IMP + NH(3). COFACTOR: Binds 1 zinc ion per subunit (By similarity). PATHWAY: Purine metabolism; IMP biosynthesis via salvage pathway; IMP from AMP: step 1/1. SUBUNIT: Homotetramer. TISSUE SPECIFICITY: Isoform 1 is the predominant form in skeletal muscle; Isoform 2 predominates in smooth muscle, non-muscle tissue, embryonic muscle and undifferentiated myoblasts; Isoform 3 is found in erythrocytes. DISEASE: Defects in AMPD3 are the cause of adenosine monophosphate deaminase deficiency erythrocyte type (AMPDDE) [MIM:612874]; also known as erythrocyte AMP deaminase deficiency. AMPDDE is a metabolic disorder due to lack of activity of the erythrocyte isoform of AMP deaminase. It is a clinically asymptomatic condition characterized by a 50% increase in steady-state levels of ATP in affected cells. Individuals with complete deficiency of erythrocyte AMP deaminase are healthy and have no hematologic disorders. SIMILARITY: Belongs to the adenosine and AMP deaminases family.
The RNAfold program from the Vienna RNA Package is used to perform the secondary structure predictions and folding calculations. The estimated folding energy is in kcal/mol. The more negative the energy, the more secondary structure the RNA is likely to have.
ModBase Predicted Comparative 3D Structure on Q01432
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Orthologous Genes in Other Species
Orthologies between human, mouse, and rat are computed by taking the best BLASTP hit, and filtering out non-syntenic hits. For more distant species reciprocal-best BLASTP hits are used. Note that the absence of an ortholog in the table below may reflect incomplete annotations in the other species rather than a true absence of the orthologous gene.