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IFI44 — PRKACB

Text-mined interactions from Literome

Salvador et al., Endocrinology 2002 : Taken together, these results indicate that, under primary culture conditions where physiological levels of signaling proteins are present, hCG signals to activate MEK, p42/44 MAPK, CREB, and histone H3 in a predominantly PKA dependent and PKC independent manner
Rius et al., Arterioscler Thromb Vasc Biol 2004 (Calcium Signaling...) : The upregulation of NOR-1 mRNA levels by LDL involves pertusis-sensitive G protein coupled receptors, Ca2+ mobilization, protein kinases A (PKA) and C ( PKC ) activation , and mitogen activated protein kinase pathways ( MAPK ) ( p44/p42 and p38 )
Whalen et al., Hum Exp Toxicol 2006 : These results indicate that AC inhibition caused alterations of the levels of key enzymes, while decreased PKA activity caused an increase in p44/42 activation
Yan et al., FASEB J 2011 : The antimitogenic efficacy of PGE ( 2 ) in inhibiting control cultures was associated with greater ability to stimulate sustained PKA activation and greater inhibition of late-phase promitogenic p42/p44 and PI3K activities