ID:CCNG1_HUMAN DESCRIPTION: RecName: Full=Cyclin-G1; Short=Cyclin-G; FUNCTION: May play a role in growth regulation. Is associated with G2/M phase arrest in response to DNA damage. May be an intermediate by which p53 mediates its role as an inhibitor of cellular proliferation (By similarity). INTERACTION: Q15025:TNIP1; NbExp=3; IntAct=EBI-3905829, EBI-357849; SUBCELLULAR LOCATION: Nucleus. Note=DNA replication foci after DNA damage. TISSUE SPECIFICITY: High levels in skeletal muscle, ovary, kidney and colon. DEVELOPMENTAL STAGE: Very low levels in normal cells during G1 phase, which increase as cells enter the S phase and stay high throughout the S and G2/M phases. In breast cancer cells consistent high levels are found throughout the cell cycle. INDUCTION: Activated in breast and prostate cancer cells. Activated by actinomycin-D induced DNA damage. SIMILARITY: Belongs to the cyclin family. Cyclin G subfamily. SEQUENCE CAUTION: Sequence=AAB03903.1; Type=Erroneous initiation; Sequence=CAA54821.1; Type=Erroneous initiation; WEB RESOURCE: Name=NIEHS-SNPs; URL="http://egp.gs.washington.edu/data/ccng1/";
The RNAfold program from the Vienna RNA Package is used to perform the secondary structure predictions and folding calculations. The estimated folding energy is in kcal/mol. The more negative the energy, the more secondary structure the RNA is likely to have.
ModBase Predicted Comparative 3D Structure on P51959
Front
Top
Side
The pictures above may be empty if there is no ModBase structure for the protein. The ModBase structure frequently covers just a fragment of the protein. You may be asked to log onto ModBase the first time you click on the pictures. It is simplest after logging in to just click on the picture again to get to the specific info on that model.
Orthologous Genes in Other Species
Orthologies between human, mouse, and rat are computed by taking the best BLASTP hit, and filtering out non-syntenic hits. For more distant species reciprocal-best BLASTP hits are used. Note that the absence of an ortholog in the table below may reflect incomplete annotations in the other species rather than a true absence of the orthologous gene.