ID:CTGF_HUMAN DESCRIPTION: RecName: Full=Connective tissue growth factor; AltName: Full=CCN family member 2; AltName: Full=Hypertrophic chondrocyte-specific protein 24; AltName: Full=Insulin-like growth factor-binding protein 8; Short=IBP-8; Short=IGF-binding protein 8; Short=IGFBP-8; Flags: Precursor; FUNCTION: Major connective tissue mitoattractant secreted by vascular endothelial cells. Promotes proliferation and differentiation of chondrocytes. Mediates heparin- and divalent cation-dependent cell adhesion in many cell types including fibroblasts, myofibroblasts, endothelial and epithelial cells. Enhances fibroblast growth factor-induced DNA synthesis. SUBUNIT: Monomer. SUBCELLULAR LOCATION: Secreted, extracellular space, extracellular matrix (By similarity). Secreted (By similarity). TISSUE SPECIFICITY: Expressed in bone marrow and thymic cells. Also expressed one of two Wilms tumors tested. SIMILARITY: Belongs to the CCN family. SIMILARITY: Contains 1 CTCK (C-terminal cystine knot-like) domain. SIMILARITY: Contains 1 IGFBP N-terminal domain. SIMILARITY: Contains 1 TSP type-1 domain. SIMILARITY: Contains 1 VWFC domain. WEB RESOURCE: Name=Atlas of Genetics and Cytogenetics in Oncology and Haematology; URL="http://atlasgeneticsoncology.org/Genes/CTGFID40192ch6q23.html";
The RNAfold program from the Vienna RNA Package is used to perform the secondary structure predictions and folding calculations. The estimated folding energy is in kcal/mol. The more negative the energy, the more secondary structure the RNA is likely to have.
Pfam Domains: PF00007 - Cystine-knot domain PF00090 - Thrombospondin type 1 domain PF00093 - von Willebrand factor type C domain PF00219 - Insulin-like growth factor binding protein PF19035 - CCN3 Nov like TSP1 domain
ModBase Predicted Comparative 3D Structure on P29279
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Orthologous Genes in Other Species
Orthologies between human, mouse, and rat are computed by taking the best BLASTP hit, and filtering out non-syntenic hits. For more distant species reciprocal-best BLASTP hits are used. Note that the absence of an ortholog in the table below may reflect incomplete annotations in the other species rather than a true absence of the orthologous gene.