Human Gene ADGRG2 (ENST00000340581.3_4) from GENCODE V47lift37
Description: Orphan receptor. Could be involved in a signal transduction pathway controlling epididymal function and male fertility. May regulate fluid exchange within epididymis. (from UniProt Q8IZP9) Gencode Transcript: ENST00000340581.3_4 Gencode Gene: ENSG00000173698.18_14 Transcript (Including UTRs) Position: hg19 chrX:19,008,128-19,100,861 Size: 92,734 Total Exon Count: 24 Strand: - Coding Region Position: hg19 chrX:19,008,982-19,086,952 Size: 77,971 Coding Exon Count: 23
ID:GPR64_HUMAN DESCRIPTION: RecName: Full=G-protein coupled receptor 64; AltName: Full=Human epididymis-specific protein 6; Short=He6; Flags: Precursor; FUNCTION: Could be involved in a signal transduction pathway controlling epididymal function and male fertility. SUBUNIT: Forms a heterodimer, consisting of a large extracellular region linked to a seven-transmembrane moiety (Probable). SUBCELLULAR LOCATION: Cell membrane; Multi-pass membrane protein. TISSUE SPECIFICITY: Epididymis specific. Both subunits were associated with apical membranes of efferent ductule and proximal epididymal duct epithelia. PTM: Proteolytically cleaved into 2 subunits, an extracellular subunit and a seven-transmembrane subunit (Potential). SIMILARITY: Belongs to the G-protein coupled receptor 2 family. LN-TM7 subfamily. SIMILARITY: Contains 1 GPS domain.
The RNAfold program from the Vienna RNA Package is used to perform the secondary structure predictions and folding calculations. The estimated folding energy is in kcal/mol. The more negative the energy, the more secondary structure the RNA is likely to have.
SCOP Domains: 81321 - Family A G protein-coupled receptor-like
ModBase Predicted Comparative 3D Structure on Q8IZP9
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Orthologous Genes in Other Species
Orthologies between human, mouse, and rat are computed by taking the best BLASTP hit, and filtering out non-syntenic hits. For more distant species reciprocal-best BLASTP hits are used. Note that the absence of an ortholog in the table below may reflect incomplete annotations in the other species rather than a true absence of the orthologous gene.