ID:WIPF2_HUMAN DESCRIPTION: RecName: Full=WAS/WASL-interacting protein family member 2; AltName: Full=WASP-interacting protein-related protein; AltName: Full=WIP- and CR16-homologous protein; AltName: Full=WIP-related protein; FUNCTION: Plays an active role in the formation of cell surface protrusions downstream of activated PDGFB receptors. Plays an important role in actin-microspike formation through cooperation with WASL. May cooperate with WASP and WASL to induce mobilization and reorganization of the actin filament system. SUBUNIT: Interacts with WASL and WASP, and this interaction results in cytoplasmic relocation of these two proteins along actin filaments. Interacts with NCK2 resulting in the localization to sites of focal adhesions. No interaction was seen with WASF2 and WASF3. INTERACTION: P62993:GRB2; NbExp=3; IntAct=EBI-2850112, EBI-401755; SUBCELLULAR LOCATION: Cytoplasm, cytoskeleton. Note=Localized to stress fibers and bundles of actin filaments. TISSUE SPECIFICITY: Expressed mainly in brain, colon, lung and stomach (at protein level). Ubiquitously expressed, with high expression in brain, kidney, lung, and placenta. MISCELLANEOUS: Access to the profilin-binding site is masked in the full-length protein. SIMILARITY: Belongs to the verprolin family. SIMILARITY: Contains 1 WH2 domain. SEQUENCE CAUTION: Sequence=AAQ15232.1; Type=Frameshift; Positions=373;
The RNAfold program from the Vienna RNA Package is used to perform the secondary structure predictions and folding calculations. The estimated folding energy is in kcal/mol. The more negative the energy, the more secondary structure the RNA is likely to have.
ModBase Predicted Comparative 3D Structure on Q8TF74
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Orthologous Genes in Other Species
Orthologies between human, mouse, and rat are computed by taking the best BLASTP hit, and filtering out non-syntenic hits. For more distant species reciprocal-best BLASTP hits are used. Note that the absence of an ortholog in the table below may reflect incomplete annotations in the other species rather than a true absence of the orthologous gene.