ID:XPP1_HUMAN DESCRIPTION: RecName: Full=Xaa-Pro aminopeptidase 1; EC=3.4.11.9; AltName: Full=Aminoacylproline aminopeptidase; AltName: Full=Cytosolic aminopeptidase P; AltName: Full=Soluble aminopeptidase P; Short=sAmp; AltName: Full=X-Pro aminopeptidase 1; AltName: Full=X-prolyl aminopeptidase 1, soluble; FUNCTION: Contributes to the degradation of bradykinin. Catalyzes the removal of a penultimate prolyl residue from the N-termini of peptides, such as Arg-Pro-Pro. CATALYTIC ACTIVITY: Release of any N-terminal amino acid, including proline, that is linked to proline, even from a dipeptide or tripeptide. COFACTOR: Binds 2 manganese ions per subunit. ENZYME REGULATION: Inhibited by apstatin and the metal ion chelators EDTA and 1,10-phenanthroline. Partially inhibited by dithiothreitol. Not inhibited by enalaprilat or amastatin. BIOPHYSICOCHEMICAL PROPERTIES: Kinetic parameters: KM=100.6 uM for bradykinin; KM=308 uM for the tripeptide Arg-Pro-Pro; pH dependence: Optimum pH is 8.2; SUBUNIT: Homodimer. SUBCELLULAR LOCATION: Cytoplasm (By similarity). TISSUE SPECIFICITY: Expressed in all tissues tested, including pancreas, heart, muscle, kidney, liver, lung and brain. Highest levels in pancreas. SIMILARITY: Belongs to the peptidase M24B family. SEQUENCE CAUTION: Sequence=CAD38640.1; Type=Erroneous initiation;
The RNAfold program from the Vienna RNA Package is used to perform the secondary structure predictions and folding calculations. The estimated folding energy is in kcal/mol. The more negative the energy, the more secondary structure the RNA is likely to have.
ModBase Predicted Comparative 3D Structure on Q9NQW7
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Orthologous Genes in Other Species
Orthologies between human, mouse, and rat are computed by taking the best BLASTP hit, and filtering out non-syntenic hits. For more distant species reciprocal-best BLASTP hits are used. Note that the absence of an ortholog in the table below may reflect incomplete annotations in the other species rather than a true absence of the orthologous gene.