Gene interactions and pathways from curated databases and text-mining

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CDC42 — CDKN1A

Protein-Protein interactions - manually collected from original source literature:

Studies that report less than 10 interactions are marked with *

Text-mined interactions from Literome

Dharmawardhane et al., Mol Biol Cell 2000 : p21 activated kinases (PAKs) are targets for activated Rac and Cdc42 guanosine 5'-triphosphatases and have been shown to regulate the actin-myosin cytoskeleton
Bao et al., Mol Cell Biol 2002 : Concomitant with our finding that the small GTPases Cdc42 and Rac1 were activated by attachment to FN, constitutively active ( ca ) Cdc42 and ca Rac1 promoted down-regulation of p21 ( CIP1 ) ... However, dominant negative ( dn ) Cdc42 and dn Rac1 mutants blocked the anchorage induced degradation of p21 ( CIP1 ), suggesting that an integrin induced Cdc42/Rac1 signaling pathway activates proteasomal degradation of p21 ( CIP1 )
Vidal et al., Blood 2002 : Cdc42/Rac1 dependent activation of the p21 activated kinase (PAK) regulates human platelet lamellipodia spreading : implication of the cortical-actin binding protein cortactin
Li et al., Gene regulation and systems biology 2008 : Of the five genes tested by quantitative RT-PCR, CDKN1A ( p21 ( waf1 ) ) was up regulated , CDC2/cdk1 , MCM6, ORC1L were down regulated, while ORC3L expression remained unchanged following butyrate treatment
Malecka et al., J Biol Chem 2013 : Autoinhibited p21 activated kinase 1 (Pak1) can be activated in vitro by the plasma membrane bound Rho GTPases Rac1 and Cdc42 as well as by the lipid phosphatidylinositol ( 4,5 ) -bisphosphate ( PIP2 )