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FADD — RALBP1

Pathways - manually collected, often from reviews:

• OpenBEL Selventa BEL large corpus: Complex of CASP8-FADD-IKBKG-RALBP1-TRAF2 → TNFSF10 (increases) Varfolomeev et al., J Biol Chem 2005*
  Evidence: Here we show that Apo2L/TRAIL activates kinase pathways by promoting the association of a secondary signaling complex, subsequent to assembly of a primary, death-inducing signaling complex (DISC). The secondary complex retained the DISC components FADD and caspase-8, but recruited several factors involved in kinase activation by TNF, namely, RIP1, TRAF2, and NEMO/IKKgamma.

Protein-Protein interactions - manually collected from original source literature:

Studies that report less than 10 interactions are marked with *

• IRef Hprd Interaction: RALBP1 — FADD (in vitro) Varfolomeev et al., J Biol Chem 2005*
• IRef Hprd Interaction: RALBP1 — FADD (in vivo) Varfolomeev et al., J Biol Chem 2005*