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UCSC Genome Browser Gene Interaction Graph
Gene interactions and pathways from curated databases and text-mining

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CNTF — MAPK6

Text-mined interactions from Literome

van Puijenbroek et al., Exp Cell Res 1999 (Carcinoma, Embryonal) : Using a dominant negative p21ras construct and a specific inhibitor of mitogen activated protein kinase kinase ( MEK ; PD098059 ) we demonstrated that CNTF induced Stat3 transactivation was independent of the p21ras-mitogen activated protein kinase ( MAPK ) pathway, while CNTF induced MAPK activation was p21ras- and MEK dependent
Peterson et al., J Neurosci 2000 : To understand its mechanism of action in vivo, the adult rat retina was used as a model to investigate CNTF mediated activation of Janus kinase/signal transducer and activator of transcription ( Jak-STAT ) and ras-mitogen activated protein kinase ( ras-MAPK )
Zvonic et al., J Biol Chem 2003 : In order to investigate the effects of CNTF on fat cells, we examined the expression of CNTF receptor complex proteins ( LIFR, gp130, and CNTFRalpha ) during adipocyte differentiation and the effects of CNTF on STAT, Akt, and MAPK activation
Chen et al., Mol Biol Cell 2005 : Furthermore, CNTF could activate the p44/p42 MAPK and down-regulate the expression of myogenic regulatory factors ( MRFs )
Crowe et al., Endocrinology 2008 (Obesity...) : Studies in cultured adipocytes revealed that CNTF activates p38 MAPK and AMP activated protein kinase
Hu et al., Hepatology 2008 (Carcinoma, Hepatocellular...) : Furthermore, CNTF induced mitogen activated protein kinase ( MAPK ) activation suppresses AMPK activity in the early phase of CNTF stimulation
Bonni et al., Science 1997 : Although CNTF stimulated both the Janus kinase-signal transducer and activator of transcription ( JAK-STAT ) and Ras-mitogen activated protein kinase signaling pathways in cortical precursor cells, the JAK-STAT signaling pathway selectively enhanced differentiation of these precursors along a glial lineage