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CRK — POT1
Protein-Protein interactions - manually collected from original source literature:
Studies that report less than 10 interactions are marked with *
Text-mined interactions from Literome
Mochizuki et al., J Biol Chem 2000
:
We have found that R-Ras, but not
Rap1 ,
mediates JNK activation by
v-Crk in 293T and NIH 3T3 cells
Posern et al., Oncogene 2000
(MAP Kinase Signaling System) :
We conclude that cellular
Crk/CRKL complexes, recruited to upstream signaling components,
contribute to basal and bombesin induced
Rap1 activity, which is independent from the Ras-Raf-Erk pathway under these circumstances
Sawada et al., J Cell Sci 2001
(MAP Kinase Signaling System) :
Rap1 is
involved in cell stretching modulation of
p38 but not ERK or JNK MAP kinase ... Furthermore, overexpression of
Rap1 enhanced
p38 activity but not ERK or JNK activity
Sakakibara et al., J Cell Sci 2002
:
Novel function of Chat in controlling cell adhesion via
Cas-Crk-C3G-pathway mediated
Rap1 activation ... Certain Cas,
Crk or C3G mutants
suppressed Rap1 activation by Chat
Ling et al., J Biol Chem 2003
(MAP Kinase Signaling System) :
Src-CrkII-C3G dependent activation of
Rap1 switches growth hormone stimulated p44/42 MAP kinase and JNK/SAPK activities
Huang et al., J Biol Chem 2004
(MAP Kinase Signaling System) :
Rap1 induced
p38 mitogen activated protein kinase activation facilitates AMPA receptor trafficking via the GDI.Rab5 complex
Hattori et al., Gan To Kagaku Ryoho 1997
(Cell Transformation, Neoplastic) :
[
Activation of
Rap1 , antagonist to ras, by
Crk-C3G ]