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IL7 — PPARA
Text-mined interactions from Literome
Borland et al., Cell Signal 2011
:
However, these studies demonstrate that stable over-expression of PPARß/d or
PPAR? significantly increases the efficacy of ligand activation and
represses UVB induced expression of tumor necrosis factor a (TNFa),
interleukin 6 (IL6), or IL8 in HaCaT keratinocytes, thereby establishing an excellent model to study the functional role of these receptors in human keratinocytes
Martín et al., J Hypertens 2012
:
In addition, pioglitazone increased the
interleukin-1ß induced
PPAR? mRNA levels