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NOS2 — TCF15

Text-mined interactions from Literome

Kim et al., Toxicol Lett 2004 : The transient expression and electrophoretic mobility shift assays with the NF-kappaB binding sites revealed that the o, p'-DDT induced increase in the iNOS and proinflammatory cytokines expression level were mediated by the NF-kappaB transcription factor
Kim et al., Biochem Biophys Res Commun 2005 : The transient expression and electrophoretic mobility shift assays revealed that the NF-kappaB transcription factor mediated the MXC induced increase in the iNOS and proinflammatory cytokines expression levels
Sheu et al., Food Chem Toxicol 2006 (Glomerulonephritis) : Contribution of conjugated linoleic acid to the suppression of inducible nitric oxide synthase expression and transcription factor activation in stimulated mouse mesangial cells
Gatti et al., J Leukoc Biol 2006 (Infection...) : Prostate epithelial cells not only express these receptors, but they are also able to respond to LPS, and LPS stimulated MAT-LU cells activate nuclear factor-kappaB transcription factor , induce the expression of inducible nitric oxide (NO) synthase , and secrete NO ... Prostate epithelial cells not only express these receptors, but they are also able to respond to LPS, and LPS stimulated MAT-LU cells activate nuclear factor-kappaB transcription factor , induce the expression of inducible nitric oxide (NO) synthase , and secrete NO
Vila-del Sol et al., J Leukoc Biol 2007 : Taken together, our data suggest that the TNF-alpha produced in response to IFN-gamma is required for iNOS induction by activating NF-kappaB transcription factor
Han et al., Biochem Biophys Res Commun 2007 (Inflammation) : The transient transfection and electrophoretic mobility shift assays with the NF-kappaB binding sites showed that the NF-kappaB transcription factor mediated the endosulfan induced increase in the expression levels of iNOS and proinflammatory cytokines
Rajapakse et al., Immunology 2008 : The data from the nuclear factor-kappaB (NF-kappaB) promoter gene transfection experiment supported the idea that inhibition of iNOS and COX-2 is caused by the down-regulation of their transcription factor , NF-kappaB
Sun et al., International journal of biological sciences 2008 : The expression of HO-1, iNOS ( Western blot ) and ICAM-1 ( cell ELISA ) proteins and activation of inflammation-relevant transcription factor , NF-kappaB ( EMSA ) were assessed
Mittal et al., Mol Cell Biochem 2011 (Respiratory Distress Syndrome, Adult) : Low environmental pH-induced iNOS gene transcription involved the activation of nuclear factor-?B ( NF-?B ) transcription factor and I?B kinases
Deans et al., J Biol Chem 1996 : Differential regulation of the two neuronal nitric-oxide synthase gene promoters by the Oct-2 transcription factor
Lin et al., J Neurochem 1997 : Regulation of astrocyte nitric oxide synthase type II expression by ATP and glutamate involves loss of transcription factor binding to DNA
Baud et al., Bull Acad Natl Med 1997 : Low environmental pH-induced iNOS gene expression involved the activation of nuclear factor-kappa B (NF-kappa B) transcription factor since [ 1 ] exposure of macrophages to low environmental pH increased NF-kappa B binding activity in the nucleus, and [ 2 ] treatment of macrophages with pyrrolidine dithiocarbamate or n-acetyl-leucinyl-norleucinal, two drugs preventing NF-kappa B translocation to the nucleus, canceled low pH-induced nitrite accumulation
Bellocq et al., J Biol Chem 1998 : Low environmental pH-induced iNOS gene transcription involved the activation of nuclear factor-kappaB (NF-kappaB) transcription factor since exposure of macrophages to low environmental pH both increased NF-kappaB binding activity in the nucleus and enhanced NF-kappaB-driven reporter gene expression
Przybocki et al., J Lab Clin Med 1998 (Hepatitis, Animal) : Additional cytokine- and oxidant mediated events such as nuclear transcription factor-kappaB activation and nitric oxide synthase induction were not observed