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FGFR3 — PLA2G1B

Text-mined interactions from Literome

Chapell et al., Toxicon 1992 : Conversely, beta-BuTX stimulated the release of ACh at a concentration ( 0.5 nM ) which caused no leakage of LDH, while N. n. atra PLA2 ( 0.5 nM ) did not stimulate ACh release ... Within 20 min, 0.5 nM beta-BuTX increased the resting release of ACh and decreased the stimulated release induced by depolarization with 4-aminopyridine, while N. n. atra PLA2 ( 0.5 nM ) did not stimulate ACh release and required 45 min to exert an inhibitory effect
Shina et al., Biochem Pharmacol 1990 : Addition of EDTA.Ca2+, which inhibits phospholipid hydrolysis, also reversed the PLA2 effect on ACh release, but not its effect on osmotic activity
Fossier et al., J Physiol 1995 : 3. Bee venom PLA2 , another monochain neurotoxic PLA2, also inhibited ACh release while non-toxic enzymatically active PLA2s like OS1 ( also purified from taipan snake venom ) or porcine pancreatic PLA2 elicited a much weaker inhibition of ACh release, suggesting a specific action of neurotoxic PLA2s versus non-toxic PLA2s on ACh release
Kan et al., Mol Pharmacol 1996 : The purposes of this study were to elucidate the type of phospholipase A2 (PLA2) involved in 6-keto-PGF1 alpha production and the mechanism ( s ) by which ACh activates PLA2 in cultured CEC