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NGF — TCF19
Text-mined interactions from Literome
Khursigara et al., J Neurosci 2001
:
The introduction of RIP2 into Schwann cells deficient in RIP2 conferred
NGF dependent nuclear
transcription factor-kappaB ( NF-kappaB ) activity and decreased the cell death induced by NGF
Cosgaya et al., J Neurochem 2001
:
Nerve growth factor (NGF) regulates the activity of the
transcription factor NF-kappaB ( nuclear factor-kappaB ) through its low affinity receptor, p75
Oh et al., J Toxicol Environ Health 2004
:
Since MAP kinase and transcription factors have been implicated in cell differentiation of neuronal cells, and our previous study demonstrated that p38 MAP kinase and AP-1 are activated during PC 12 cell differentiation, the effect of OTA on
NGF induced p38 MAP kinase and
transcription factor activation was examined
Ahamed et al., J Immunol 2004
:
Furthermore, C3a enhances
NGF induced
transcription factor activation and chemokine production via a G protein mediated pathway that does not involve TrkA phosphorylation
Kimura et al., J Cell Biol 1992
:
In addition, both SDGF and
NGF induce the
transcription factor , NGFI-A
McCauslin et al., J Biol Chem 2006
(Glioma) :
We previously showed that the
transcription factor CCAAT/enhancer binding protein delta ( C/EBPdelta )
contributes to
NGF gene regulation
Su et al., J Neurosci Res 2007
:
We conclude that preferential activation of
transcription factor binding at this AP-1 site
mediates preferential
NGF activation of the UPAR gene
Takei et al., Mol Biol Cell 2008
(Neuroblastoma...) :
We find that
NGF can
induce TNF-alpha synthesis through the nuclear factor-kappaB
transcription factor
Leugers et al., J Biol Chem 2010
(MAP Kinase Signaling System) :
In addition, we demonstrate that Tau potentiates AP-1
transcription factor activation in
response to
nerve growth factor (NGF)
Li et al., PloS one 2011
(Atrophy) :
Egr3 is a
transcription factor regulated by
NGF signaling in sympathetic neurons that is essential for normal SNS development
deSouza et al., J Mol Neurosci 1997
:
On the other hand, expression of a mutated form of this
transcription factor lacking the DNA binding domain
attenuated NGF responsiveness, without affecting basal levels of expression
Furukawa et al., J Neurosci Res 1998
:
Presenilin-1 mutation alters
NGF induced neurite outgrowth, calcium homeostasis, and
transcription factor ( AP-1 ) activation in PC12 cells