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PRKAR1A — TCF19

Text-mined interactions from Literome

Hino et al., Mol Cell Biol 2005 : Ser675 was found to be a site for phosphorylation by PKA, and substitution of this serine residue with alanine in beta-catenin attenuated inhibition of the ubiquitination of beta-catenin by PKA, PKA induced stabilization of beta-catenin, and PKA dependent activation of Tcf
Ponsioen et al., Exp Cell Res 2007 : This degree of cAMP transfer sufficed to evoke a well characterized response to cAMP in neighbor cells, i.e. the PKA mediated phosphorylation of the ER transcription factor in A431 carcinoma cells
O'Driscoll et al., J Neurochem 2007 : Activation of the transcription factor cAMP-response element binding protein ( CREB ) and subsequent up-regulation of Bcl-2 in response to bFGF was also dependent on PKA as inhibition with H-89 attenuated increased pCREB levels and Bcl-2 expression
Taurin et al., Am J Physiol Cell Physiol 2008 : This translates to a PKA dependent stimulation of TCF transcriptional activity through an increased association of phosphorylated ( by PKA ) beta-catenin with TCF-4
Ren et al., Genomics 2009 : Mutagenesis and cotransfection experiments showed that PKA regulation of this c-fos intronic element was mediated by two adjacent CRE-like sequences and the transcription factor CREB
Oeseburg et al., Arterioscler Thromb Vasc Biol 2010 (Diabetes Mellitus...) : Further analysis revealed that GLP-1 activates the cAMP response element binding ( CREB ) transcription factor in a cAMP/protein kinase A (PKA) dependent manner, and inhibition of the cAMP/PKA pathway abolished the GLP-1 protective effect
Yen et al., J Biol Chem 2011 (MAP Kinase Signaling System) : PKA , PI3K, and ERK inhibitors abolished PGE2- and cAMP induced c-Fos and MMP-9 up-regulation, and ERK activation was required for the binding of activator protein 1 (AP-1) transcription factor to the MMP-9 promoter
Su et al., PloS one 2012 : In the present studies, we have demonstrated that PKA enhances NF-E2 SUMOylation in an in vitro system using purified proteins, suggesting a possible mechanism for PKA dependent activation of the NF-E2 transcription factor through SUMOylation
Hagiwara et al., Mol Cell Biol 1993 : Cyclic AMP ( cAMP ) regulates a number of eukaryotic genes by mediating the protein kinase A (PKA) dependent phosphorylation of the CREB transcription factor at Ser-133
Richards et al., J Biol Chem 1996 : The transcription factor CREB ( cAMP responsive element binding protein ) is activated by protein kinase A (PKA) phosphorylation of a single serine residue
Jaspers et al., J Cell Physiol 1998 (Adenocarcinoma, Bronchiolo-Alveolar...) : Exposure to ozone increased interleukin 8 expression and transcription factor activities in a protein tyrosine kinase ( PTK ) -dependent and protein kinase A (PKA) dependent , yet protein kinase C ( PKC ) -independent, manner
Zanger et al., Mol Endocrinol 1999 : The pituitary-specific transcription factor , Pit-1, is necessary to mediate protein kinase A (PKA) regulation of the GH, PRL, and TSH-beta subunit genes in the pituitary