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GRAP2 — STAT3

Text-mined interactions from Literome

Turkson et al., Mol Cell Biol 1999 (Cell Transformation, Neoplastic...) : Requirement for Ras/Rac1 mediated p38 and c-Jun N-terminal kinase signaling in Stat3 transcriptional activity induced by the Src oncoprotein
Uddin et al., J Biol Chem 2000 : In studies to determine whether the regulatory effects of p38 are mediated by serine phosphorylation of Stat1 or Stat3, we found that the p38 kinase inhibitors SB203580 or SB202190 or overexpression of a dominant negative p38 mutant do not inhibit phosphorylation of Stat1 or Stat3 on Ser-727 in several IFNalpha-sensitive cell lines
Tarn et al., J Virol 2002 (Cell Transformation, Neoplastic) : Employing dominant negative mutants and inhibitors of mitogenic pathways, we demonstrate that maximum, pX-dependent Stat3 transactivation is inhibited by the p38 mitogen activated protein kinase ( MAPK ) -specific inhibitor SB 203580
Park et al., J Invest Dermatol 2008 (Keloid) : The level of Smad2/3, signal transducer and activator of transcription-3 ( STAT3 ), and p38 phosphorylation is more enhanced in KFs, and EGCG inhibited phosphorylation of phosphatidylinositol-3-kinase (PI3K), extracellular signal regulated protein kinase 1/2 ( ERK1/2 ), and STAT3 ( Tyr705 and Ser727 )
Schaljo et al., J Immunol 2009 (Inflammation) : We found that IL-10 initiates a Stat3 dependent increase of TTP expression accompanied by a delayed decrease of p38 MAPK activity