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CALM1 — MAPK15

Text-mined interactions from Literome

Naidu et al., Am J Physiol 1999 : Calcium-calmodulin mediates bradykinin induced MAPK phosphorylation and c-fos induction in vascular cells
Liu et al., J Neurosci 1999 : Differential roles of Ca ( 2+ ) /calmodulin dependent protein kinase II and mitogen activated protein kinase activation in hippocampal long-term potentiation ... The roles of Ca ( 2+ ) /calmodulin dependent protein kinase II ( CaM kinase II ) and mitogen activated protein kinase ( MAPK ) in long-term potentiation ( LTP ) were investigated in the CA1 area of hippocampal slices, using electrophysiological and biochemical approaches
Yan et al., Proc Natl Acad Sci U S A 1999 : D ( 2 ) receptor induced CREB phosphorylation was mediated by activation of protein kinase C and Ca ( 2+ ) /calmodulin dependent protein kinase, but not MAPK
Onofri et al., J Biol Chem 2000 : Phosphorylation of synapsin I in the COOH-terminal region by Ca ( 2+ ) /calmodulin dependent protein kinase II or mitogen activated protein kinase modulated the binding to the SH3 domains of amphiphysins I/II, Crk, and alpha-spectrin without affecting the high affinity interactions
Ferrer et al., J Neural Transm 2001 (Alzheimer Disease...) : Calcium/calmodulin dependent kinase II ( alpha- and beta-CaM kinase II ), and phosphorylated mitogen activated extracellular signal regulated protein kinase ( MAPK/ERK-P ), phosphorylated protein kinase of 38 kDa ( p38-P ) and phosphorylated stress activated protein kinase ( SAPK/JNK-P ) expression have been examined in Alzheimer disease (AD), Pick's disease (PiD), progressive supranuclear palsy ( PSP ) and corticobasal degeneration (CBD)
Verploegen et al., Eur J Biochem 2002 (Calcium Signaling) : Inhibition of calmodulin using a calmodulin antagonist N- ( 6-aminohexyl ) -5-chloro-1-naphthalenesulfonamide ( W7 ), did not effect N-formyl-methionyl-leucyl-phenylalanine ( fMLP ) induced ERK, p38 MAPK or GSK-3alpha phosphorylation, but attenuated fMLP induced PKB phosphorylation
Fueta et al., Life Sci 2002 : Hyperexcitability and changes in activities of Ca2+/calmodulin dependent kinase II and mitogen activated protein kinase in the hippocampus of rats exposed to 1-bromopropane ... We studied whether the hyperexcitability is associated with changes of Ca2+/calmodulin dependent kinase II ( CaMKII ), mitogen activated protein kinase ( MAPK ), and protein kinase C ( PKC )
Hilgenberg et al., J Neurobiol 2004 : Ca2+ release from intracellular stores activates both calcium/calmodulin dependent kinase II ( CaMKII ) and mitogen activated protein kinase ( MAPK )
Tang et al., J Neurosci 2005 (Calcium Signaling...) : Using techniques to overexpress or suppress kinase activity, we find that calcium/calmodulin dependent protein kinase II ( CaMKII ) and mitogen activated protein kinase ( MAPK ) are major downstream targets of the netrin-1 calcium signaling pathway and are required for axon branching
Kumada et al., J Neurosci 2006 (Disease Models, Animal...) : Each second messenger has multiple but distinct downstream targets, including Ca2+/calmodulin dependent protein kinase II, calcineurin, protein phosphatase 1, Rho GTPase, mitogen activated protein kinase , and phosphoinositide 3-kinase
Bourguignon et al., J Biol Chem 2006 (Carcinoma, Squamous Cell...) : Overexpression of the LARG-PDZ domain also functions as a dominant negative mutant ( similar to the PLC/Ca2+-calmodulin dependent kinase II ( CaMKII ) and EGFR/MAPK inhibitor effects ) to block HA/CD44 mediated signaling events ( e.g. EGFR kinase activation, Ras/RhoA co-activation, Raf-ERK signaling, PLC epsilon mediated inositol 1,4,5-triphosphate production, intracellular Ca2+ mobilization, CaMKII activity, filamin phosphorylation, and filamin-actin binding ) and to abrogate tumor cell growth/migration
Zhang et al., J Physiol 2007 : The effect was blocked by alpha7-nAChR antagonists, by chelation of intracellular Ca2+ with BAPTA, and by inhibition of both Ca2+-calmodulin dependent protein kinase II and mitogen activated protein kinase
Boddupalli et al., Free Radic Biol Med 2007 : Nitric oxide inhibits interleukin-12 p40 through p38 MAPK mediated regulation of calmodulin and c-rel
Mao et al., Brain Res Bull 2007 : Interestingly, the PMA induced CREB phosphorylation was also blocked by a calcium/calmodulin dependent protein kinase inhibitor KN93 and the two mitogen activated protein kinase ( MAPK ) kinase inhibitors PD98059 and U0126, but not by a p38 MAPK inhibitor SB203580
Shin et al., J Biol Chem 2007 (Ion Channel Gating) : Inhibition of calcium/calmodulin dependent protein kinase II activity, but not p38 MAPK , also redistributed HCN1 in CA1 pyramidal neurons
Arredondo et al., FASEB J 2008 : Cumulative results indicated that the alpha3 ( beta2/beta4 ) to alpha3 ( beta2/beta4 ) alpha5 nAChR transition predominantly involved protein kinase C, alpha3 ( beta2/beta4 ) alpha5 to alpha7 nAChR transition-Ca ( 2+ ) /calmodulin dependent protein kinase II and p38 MAPK , and alpha7 self-up-regulation-the p38 MAPK/Akt pathway, and JAK-2
Wang et al., J Biol Chem 2009 (Carcinoma, Hepatocellular) : Here we report that melittin can induce apoptosis of HCC cells by activating Ca2+/calmodulin dependent protein kinase, transforming growth factor-beta activated kinase 1 ( TAK1 ), and JNK/p38 MAPK
Moretó et al., Cell Signal 2009 (MAP Kinase Signaling System) : Differential involvement of H- and K-Ras in Raf-1 activation determines the role of calmodulin in MAPK signaling
Liang et al., J Leukoc Biol 1995 : These kinases include calcium/calmodulin dependent protein kinase II ( CAMPKII ), mitogen activated protein kinase ( MAPK ), and histone H4 protein kinase ( PKH4 )
Eguchi et al., J Biol Chem 1996 : The calmodulin inhibitor, calmidazolium, and the tyrosine kinase inhibitor, genistein, completely blocked MAPK activation by Ang II as well as by the Ca2+ ionophore A23187
Suzuki et al., Nihon Shinkei Seishin Yakurigaku Zasshi 1996 (Synaptic Transmission) : I review in this article the two groups of PSD proteins : Protein kinases ( Ca2+/calmodulin dependent protein kinase II, protein kinase C, mitogen activated protein kinase , and tyrosine kinases ) and molecules that work as messengers from the synapse to the nucleus ( MSNs )
Muthalif et al., J Biol Chem 1996 : We have investigated the contribution of Ca2+/calmodulin dependent protein kinase II ( CaM kinase II ) and mitogen activated protein kinase ( MAP kinase ) in norepinephrine ( NE ) -induced arachidonic acid ( AA ) release in rabbit aortic vascular smooth muscle cells ( VSMC )
Della Rocca et al., J Biol Chem 1997 : Chemical inhibitors of calmodulin , but not of PKC, and overexpression of a dominant negative mutant of the protein-tyrosine kinase Pyk2 also attenuate mitogen activated protein kinase phosphorylation after both alpha1B- and alpha2A-AR stimulation