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HDAC3 — NCOA1

Pathways - manually collected, often from reviews:

• Reactome Reaction: HDAC3 → NCOA1 (reaction) Gelman et al., J Biol Chem 1999, Desvergne et al., Endocr Rev 1999, Wolfrum et al., Proc Natl Acad Sci U S A 2001, Qi et al., Cell Biochem Biophys 2000, Perissi et al., Cell 2004, Feige et al., Prog Lipid Res 2006, Gouni-Berthold et al., Curr Drug Targets Cardiovasc Haematol Disord 2005, Mochizuki et al., Life Sci 2006, Farmer et al., Cell Metab 2006, Yu et al., Biochim Biophys Acta 2007, Kersten et al., PPAR research 2008, Hostetler et al., J Lipid Res 2009, Lambe et al., Eur J Biochem 1996, Lemberger et al., Annu Rev Cell Dev Biol 1996, Mukherjee et al., J Biol Chem 1997, Krey et al., Mol Endocrinol 1997

Protein-Protein interactions - manually collected from original source literature:

Studies that report less than 10 interactions are marked with *

• IRef Hprd Interaction: HDAC3 — NCOA1 (in vivo) Fajas et al., Dev Cell 2002*

Text-mined interactions from Literome

Sohn et al., Mol Endocrinol 2003 : First, corepressor SMRT [ for silencing mediator of thyroid hormone receptor ( TR ) and retinoic acid receptor ( RAR ) ] inhibits the interaction of coactivator steroid receptor coactivator-1 with liganded TR/RAR
Ishii et al., Mol Endocrinol 2004 (Thyroid Hormone Resistance Syndrome) : TR and NCoR were located on the promoter, and T3 caused NCoR dissociation and steroid receptor coactivator-1 recruitment