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ITGAM — MAPK3

Text-mined interactions from Literome

Forsberg et al., Immunology 2001 : Taken together, these results showed that TNF-alpha potentiates the CR3 mediated respiratory burst in neutrophils not only by triggering a p38 MAPK dependent up-regulation of CD11b/CD18 but also by modulating the signalling pathways
Suzuki et al., J Immunol 2001 (MAP Kinase Signaling System) : IL-1beta also induced O ( 2 ) ( - ) release and up-regulation of CD11b and CD15, and both responses were inhibited by SB203580 ( p38 MAPK inhibitor ), suggesting that p38 MAPK activation mediates IL-1beta induced O ( 2 ) ( - ) release and up-regulation of CD11b and CD15
Wong et al., Am J Respir Cell Mol Biol 2003 : IL-3, IL-5, and GM-CSF could enhance p38 MAPK and NF-kappaB activity and induce ICAM-1, CD11b , and CD18 expressions on eosinophils
Elbim et al., Clin Diagn Lab Immunol 2005 : In addition, genistein, a tyrosine kinase inhibitor, and SB203580, a p38 mitogen activated protein kinase ( p38MAPK ) inhibitor, completely reversed the decreased level of fMLP binding and increased the level of CD11b expression after IL-18 treatment
Ghori et al., Eur J Anaesthesiol 2010 (Reperfusion Injury) : This decrease in p38 MAPK activation results in decreased neutrophil CD11b/CD18 molecule expression