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CCL24 — IL13

Text-mined interactions from Literome

Lezcano-Meza et al., Allergy 2003 (Nasal Polyps) : Interleukin (IL)-4 and to a lesser extent either IL-13 or interferon-gamma regulate the production of eotaxin-2/CCL24 in nasal polyps
Yang et al., J Allergy Clin Immunol 2003 (Bronchial Hyperreactivity...) : Eotaxin-2 and IL-5 cooperatively promoted eosinophil accumulation, IL-13 production, and AHR to methacholine ... IL-13 production was critically dependent on eotaxin-2- and IL-5 regulated eosinophilia, which predisposed to the development of AHR
Kobayashi et al., Ann Allergy Asthma Immunol 2004 : Eotaxin-1 and eotaxin-3, but not eotaxin-2 , mRNA expressions were induced by stimulation with interleukin (IL) 13 or IL-4
Kagami et al., Clin Exp Immunol 2005 (Dermatitis, Atopic) : The production of CCL24 in HaCaT cells was slightly enhanced by IL-4 and that of CCL26 was strongly enhanced by IL-4 and IL-13
van Wetering et al., Mol Immunol 2007 : Epithelial differentiation is a determinant in the production of eotaxin-2 and -3 by bronchial epithelial cells in response to IL-4 and IL-13
Wu et al., Clin Exp Allergy 2009 (Asthma...) : Primary alveolar macrophages ( pAMs ) from naïve BALB/c mice were cultured to determine whether Mp-induced PGE ( 2 ) or exogenous PGE ( 2 ) down-regulates IL-4/IL-13 induced eotaxin-2 ... In pAMs, IL-4/IL-13 significantly increased eotaxin-2 , which was reduced by Mp infection accompanied by dose dependent PGE ( 2 ) induction ... Exogenous PGE ( 2 ) inhibited IL-4/IL-13 induced eotaxin-2 in a dose dependent manner
Neilsen et al., Clin Exp Allergy 2010 : Interleukin-13 directly promotes oesophagus production of CCL11 and CCL24 and the migration of eosinophils ... Our data demonstrate that IL-13 is a potent and direct inducer of both CCL11 and CCL24 production from the oesophagus, as is IL-4 also
Crapster-Pregont et al., J Allergy Clin Immunol 2012 (Inflammation) : Dendritic cell and alveolar macrophage depletion protected mice from IL-13 induced airway inflammation and CCL11, CCL24 , CCL22, and CCL17 chemokine production ... Preferential depletion of dendritic cells protected mice from IL-13 induced airway inflammation and CCL22 and CCL17 chemokine production but not from IL-13 induced CCL11 and CCL24 chemokine production