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IL7 — SELE

Text-mined interactions from Literome

Cockerill et al., Circulation 2001 (Acute Disease...) : Elevation of plasma high-density lipoprotein concentration reduces interleukin-1 induced expression of E-selectin in an in vivo model of acute inflammation
Leeuwenberg et al., Eur J Immunol 1991 (Leukemia, Myeloid) : ELAM-1 expression can selectively be induced on human umbilical vein endothelial cells ( HUVEC ) by tumor necrosis factor, interleukin 1 and lipopolysaccharide
Lewis et al., Mol Cell Biol 1994 : In addition, an essential role for cooperative interaction between the two NF-kappa B complexes is shown by the requirement for both NF-kappa B sites to mediate E-selectin promoter activation by interleukin-1 and p50/p65 expression
Weston et al., Transpl Immunol 1995 (Kidney Failure, Chronic) : E-Selectin is a 115-kDa cell surface glycoprotein transiently expressed on vascular endothelium in response to interleukin-1 and tumour necrosis factor-alpha with a peak in expression at four hours
Ray et al., Biochem J 1997 : Induction of the E-selectin promoter by interleukin 1 and tumour necrosis factor alpha, and inhibition by glucocorticoids
Miller et al., J Leukoc Biol 1998 : Utilizing neutralizing antibodies, we show that CD40L mediated tissue factor and thrombomodulin modulation, as well as E-selectin and VCAM-1 upregulation, is independent of tumor necrosis factor alpha, interleukin-1alpha , or interleukin-1beta production
Friedrichs et al., Arterioscler Thromb Vasc Biol 1998 : Inhibition of tumor necrosis factor-alpha- and interleukin-1 induced endothelial E-selectin expression by thiol modifying agents