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FGFR4 — STAT1

Protein-Protein interactions - manually collected from original source literature:

Studies that report less than 10 interactions are marked with *

• IRef Hprd Interaction: FGFR4 — STAT1 (in vitro) Hart et al., Oncogene 2000, Hart et al., Mol Biol Cell 2001, Uddin et al., J Biol Chem 2002, Dimberg et al., Blood 2003*, Deb et al., J Immunol 2003, DeVries et al., J Biol Chem 2004, Wen et al., Cell 1995*, Quelle et al., J Biol Chem 1995, Shuai et al., Science 1993
• IRef Hprd Interaction: FGFR4 — STAT1 (in vivo) Hart et al., Oncogene 2000, Hart et al., Mol Biol Cell 2001, Uddin et al., J Biol Chem 2002, Dimberg et al., Blood 2003*, Deb et al., J Immunol 2003, DeVries et al., J Biol Chem 2004, Wen et al., Cell 1995*, Quelle et al., J Biol Chem 1995, Shuai et al., Science 1993

Text-mined interactions from Literome

Britto et al., Cleft Palate Craniofac J 2002 (Acrocephalosyndactylia...) : In addition, the immunolocalization of the ligand FGFs 2, 4, and 7 was undertaken together with the intracellular transcription factor STAT1 , which is activated by FGFR signaling
Citores et al., J Cell Physiol 2007 (Breast Neoplasms) : Both FGFR1 and FGFR2 induced strong phosphorylation of STAT1 causing redistribution of the Golgi apparatus, while FGFR3 and FGFR4 induced less phosphorylation of STAT1 and little or no redistribution of the Golgi apparatus
Su et al., Nature 1997 (Thanatophoric Dysplasia) : Activation of Stat1 by mutant fibroblast growth-factor receptor in thanatophoric dysplasia type II dwarfism