◀ Back to IL16
CASP14 — IL16
Text-mined interactions from Literome
Ludwiczek et al., Eur Cytokine Netw 2001
:
Activation of
caspase-3 by interferon alpha
causes interleukin-16 secretion but fails to modulate activation induced cell death
Reich et al., J Immunol 2001
(Dermatitis, Atopic) :
There was evidence that LLDC use caspase-1 to process IL-16, as inhibition of
caspase-1 , but not of caspase-3, partially
prevented the release of
IL-16 in response to ligation of FcepsilonRI
Gianoukakis et al., Endocrinology 2003
:
IL-16 but not RANTES expression was
dependent on the activity of
caspase-3
Guo et al., Blood 2003
:
In the present study we confirm that Fas signaling induces both phenotypic and functional maturation of murine DCs, and we demonstrate that phenotypic maturation is associated with phosphorylation of extracellular signal regulated kinase ( ERK ) 1/2,
activation of
caspase-1 , and secretion of
interleukin-beta ( IL-1beta )
Elssner et al., J Immunol 2004
:
In contrast, stimulation with bacterial LPS inhibited
caspase-3 activation and significantly
inhibited the release of
IL-16
Skundric et al., J Autoimmun 2005
(Encephalomyelitis, Autoimmune, Experimental...) :
Our data suggest that
caspase-3 mediated production of
IL-16 by infiltrating CD4+ T cells, contributes to ongoing neuroinflammation by chemoattraction of additional waves of CD4+ T cells
Wang et al., Ann Thorac Surg 2005
(Reperfusion Injury) :
Myocardial tissue was then assessed for products of p38 MAPK activation, expression of tumor necrosis factor,
interleukin-1beta and interleukin-6, and
activation of caspase-1,
caspase-3 and caspase-11
Shushanov et al., Bull Exp Biol Med 2010
:
Insulin-like growth factor-1 and insulin inhibit
caspase-3/7 activation
initiated by
interleukin-1ß in the rat eye retinal neurons
Liao et al., Inflamm Res 2013
:
Lipopolysaccharide/adenosine triphosphate mediated signal transduction in the regulation of NLRP3 protein expression and
caspase-1 mediated
interleukin-1ß secretion