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CRH — MSH5

Text-mined interactions from Literome

Fekete et al., Neurosci Lett 2000 : Whereas fasting suppressed CRH mRNA levels in the PVN, alpha-MSH at doses of 150 and 300 ng every 6 h for 64 h prevented the fasting induced suppression of CRH gene expression in the PVN ... These data indicate that the suppression of alpha-MSH synthesis may be responsible for the decreased CRH gene expression in the PVN during fasting
Vecsernyés et al., Endocr Res 2000 : Our results reveal that both the anxiogenic and the PAA activating effects of alpha-MSH are mediated by CRF
Xia et al., J Neuroimmunol 2001 (Bacterial Infections...) : In the hypothalamic paraventricular nucleus, a major neuroendocrine and autonomic center, gamma ( 2 ) -MSH inhibits LPS induced increases in CRF mRNA levels, but does not suppress LPS augmented arginine vasopressin heteronuclear RNA expression
Sarkar et al., Brain Res 2002 : As alpha-MSH also induces the activation of CRH gene expression in the PVN, we further determined whether i.c.v. injection of alpha-MSH regulates the phosphorylation of CREB in hypophysiotropic CRH neurons ... These data demonstrate that i.c.v. alpha-MSH administration increases the phosphorylation of CREB in several subdivisions of the PVN including TRH and CRH neurons in the medial and periventricular parvocellular subdivisions, suggesting that phosphorylation of CREB may be necessary for alpha-MSH induced activation of the TRH and CRH genes
Van der Salm et al., Gen Comp Endocrinol 2004 : The release of alpha-MSH was stimulated by TRH and CRH , and was inhibited by MCH and DA
van den Burg et al., Gen Comp Endocrinol 2005 : The origin of this variable response was investigated by ( 1 ) studying the effects of corticotropin releasing hormone (CRH) on alpha-MSH and NAc beta-end release in vitro, ( 2 ) establishing where in the second messenger pathway coupled to CRH receptors melanotrope responsiveness is determined, and ( 3 ) testing modulatory actions of other hypothalamic factors ( here opioid beta-endorphin )
Flik et al., Gen Comp Endocrinol 2006 : Endorphin, produced in the NPO and transported via axons to the pituitary gland in vivo, reverses the stimulatory CRF action on MSH-cells to a differential inhibition of N-acetyl beta-endorphin release in vitro ( MSH release is not affected )
Catania et al., Brain Res Bull 1991 : Similarly, CRH ( 1 and 10 micrograms/kg IV ) did not affect plasma alpha-MSH , whereas it stimulated ACTH and corticosterone release
Sobel et al., Endocr Res 1990 : Bacterial lipopolysaccharide (LPS) and corticotropin releasing hormone (CRH) plus arginine vasopressin (AVP) induce immunoassayable ( 1-13 ) ACTH ( alpha MSH ) from mononuclear leukocytes
Tachibana et al., Comp Biochem Physiol A Mol Integr Physiol 2007 (Anorexia) : Therefore, we investigated whether the anorexic effect of alpha-MSH is mediated by corticotrophin releasing factor (CRF) in the domestic chick
van Oers et al., Endocrinology 1989 : Finally, we found that PFU 83 did not affect resting or ether induced alpha MSH secretion, indicating that CRF does not play a major role in the control of alpha MSH secretion
Kemppainen et al., Endocrinology 1989 : Cortisol had no effect on either basal or CRF stimulated secretion of IR-alpha MSH or IR-ACTH from dog PI cells, but it did inhibit CRF stimulated IR-ACTH from perifused dog PD
Verburg-Van Kemenade et al., Peptides 1987 : During in vitro superfusion of neurointermediate lobe tissue, CRF , sauvagine and urotensin I induced a rapid and dose dependent stimulation of secretion of MSH and endorphin
Castro et al., Comp Biochem Physiol C 1988 : 4. AVT and CRF when perfused together acted synergistically on alpha-MSH release yielding a dose response line whose slope approximated that of DME
Hotta et al., Regul Pept 1988 : CRH stimulated not only IR-beta-Ep and IR-adrenocorticotropic hormone ( ACTH ) but also IR-alpha-melanocyte stimulating hormone ( IR-alpha-MSH ) secretions from fetal pituitaries
Fazekas et al., Exp Clin Endocrinol 1984 : Synthetic ovine CRF stimulated the release of both immunoreactive ACTH and bioactive alpha-MSH in dose dependent fashion
Meunier et al., Can J Biochem Cell Biol 1983 : A requirement for calcium ions was observed on basal as well as on CRF induced alpha-MSH secretion
Tozawa et al., Neurosci Lett 1994 : These results suggest that there is a short negative feedback effect of alpha MSH on CRF release and it appears only in the absence of a long negative feedback effect of glucocorticoids
Colao et al., Horm Res 1996 (Cushing Syndrome) : No difference in sensitivity to dexamethasone ( urinary cortisol percent decrease : 66.4 +/- 4.9 vs. 67.8 +/- 3.4 ) and surgery outcome ( chi 2 test : p = 0.7 ) was found between patients with alpha-MSH response to CRH and patients without such a response ... By contrast, baseline PRL levels, although being normal in both groups, were significantly higher in patients with alpha-MSH response to CRH ( 18.1 +/- 1.6 vs. 10.1 +/- 0.7 micrograms/l ; p < 0.001 )