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IL8 — PRR9

Text-mined interactions from Literome

Zhong et al., J Biol Chem 2007 (MAP Kinase Signaling System) : The JNK and p38 MAPK groups are major PRR effectors and are key to the PRR dependent induction and release of proinflammatory cytokines such as tumor necrosis factor and interleukin-8
Buchholz et al., Infect Immun 2008 (Inflammation) : The role of PRR such as Toll-like receptors ( TLR ) and nucleotide binding oligomerization domain ( NOD ) proteins in the endogenous interleukin-8 (IL-8) response induced during Chlamydia trachomatis infection is not known ... We hypothesized that a PRR is essential for the IL-8 response induced by C. trachomatis infection ... We conclude that the endogenous IL-8 response induced by C. trachomatis infection is dependent upon NOD1 PRR signaling through RIP2 as part of a signal system requiring multiple inputs for optimal IL-8 induction
Zubcevic et al., Hypertension 2013 (Hypertension) : Finally, PRR activation in SHR neuronal cultures by prorenin activated nuclear factor-?B and increased mRNA levels of interleukin-1ß ( 250-fold ), tumor necrosis factor-a ( 32-fold ), interleukin-6 ( 35-fold ), C-C motif ligand 5 ( 12-fold ), and interleukin-10 ( 7-fold ) in a nuclear factor-?B dependent but angiotensin II type 1 receptor independent manner